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Its neuroprotective potential lies in its ability to target multiple signaling pathways involving AMP-activated protein kinase (AMPK), neurotrophins such as brain-derived neurotrophic factor (BDNF), transcription factors such as cAMP response element-binding protein (CREB), nuclear factor kappa-light-chain-enhancer of activated B cells (NF-B), nuclear factor erythroid 2-related factor 2 (Nrf-2), and others

These results imply that sulforaphane may be able to prevent oxidative damage in the brain after rotenone exposure

10.1038/s41598-018-19573-x 23 CaoY

Wolf BB, Schuler M, Echeverri F, Green DR

doi: 10.1007/s00232-014-9745-x 132

REM stays intact, no GABA-A clamp, no dependence documented in human trials