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Accumulating clinical and experimental evidence confirms ferroptosis as a pivotal driver of kidney disease onset and progression

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1 1 mmHg, P = 0006), backward pressure (Pb) ( 3 1 v

Protons are allowed to flow back into the matrix through a molecular turbine known as ATP synthase (complex V), which harnesses the proton motive force to phosphorylate ADP to ATP [37]

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Therefore, it leads to the activation of several compensatory mechanisms by the maternal system to maintain glucose homeostasis during this stage