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Evidence is provided that asymptomatic meningococcal carriage on the nasopharyngeal mucosal surface is facilitated by localized hypermutation in contingency genes, which, together with horizontal gene transfer, affects the expression of a plethora of surface determinants involved in meningococcal adhesion, metabolism, intracellular survival, intra-species and interspecies competition, and escape of immune response (Green et al., 2020)

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Interestingly, improved supply of GSH precursors from astrocytes to neurons and a thereby better resistance of neurons has been reported after adrenergic stimulation (Yoshioka et al