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These findings seem to parallel the inhibitory effects of bilirubin on superoxide production32 and on overall mitochondrial metabolism.33 In fact, pyrrole groups of bilirubin, present also in our compounds, have been suggested to compete with NADH on the active sites of mitochondrial dehydrogenases indicating possible mechanisms of these effects.33 This conclusion is also in accord with a report by Boloor, et al., who described marked protective effects of chlorophyllin on mitochondria-induced lipid peroxidation.34 It seems that observed anti-carcinogenic effects are mediated, at least partially, by direct inhibition of mitochondrial ROS generation

10.1038/s41423-022-00905-x 203 WeissT.WellerM.RothP

In particular, levels of L-arginine, L-proline, and L-glutamic acid were elevated, whereas glutathione, L-tyrosine, and L-phenylalanyl-L-proline levels were reduced

The CY 2026 OPPS/ASC proposed rule inadvertently stated the proposed CY 2026 ASC weight scalar was 0.842 (90 FR 33752 and 90 FR 33846)

S2CID 8959463

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