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Ho PI, Collins SC, Dhitavat S, Ortiz D, Ashline D, Rogers E, Shea TB

Figure 3 4.2.2 Macrophages In RA, activity imbalance between pro-inflammatory M1 macrophages and anti-inflammatory M2 macrophages elicits inflammation and autoimmunity in the synovium, leading to joint damage ( 2+ can enter mitochondria to maintain mitochondrial homeostasis ( 2+ levels, which promoted the phosphorylation of serum response factor (SRF) and its binding to the vascular endothelial growth factor receptor 2 (VEGFR2) promoter, producing VEGFR2 and thereby promoting inflammatory angiogenesis

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After TBI, there is an activation of neuroinflammation response pathway mediated by microglia cells that were not initially damaged, and the activated microglial cells may promote the release of proinflammatory factors such as interleukin 1, tumor necrosis factor, nitric oxide, etc., (Kumar and Loane, 2012), inducing a progressive cycle of chronic inflammation (Sulhan et al., 2020)

Xie D, Wang P, Chen W, Lin J, Wu M, Wang Y, et al