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Each has documented limitations: NSAIDs: Reduce inflammation short-term but inhibit prostaglandin pathways needed for tissue repair

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For example, the gel-to-liquid crystalline phase transition temperature (Tm) for an ether-linked phosphatidylcholine (1,2-dihexadecyl-sn-glycero-3-phosphocholine) is higher than its ester-linked analog (1,2-dipalmitoyl-sn-glycero-3-phosphocholine), though specific numerical values for the dihexadecylglycerol backbone are not directly reported [1]

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This potentially suggests that BPC-157 may activate the FAK-paxillin pathway, which may be involved in the promotion of tendon fibroblast migration and cell adhesion

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