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Damaged TECs promote inflammation and fibrosis through several mechanisms, including EMT, the release of pro-inflammatory factors, and the activation of signaling pathways, such as the TGF-1/Smad3 pathway ( 3.2.2 Ferroptosis-inflammation-fibrosis Ferroptosis not only directly affects the survival of renal TECs but also affects the inflammatory response by inducing macrophage polarization

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References Sourced from Neelakantan et al., Biochemical Pharmacology (2018), on NNMT inhibitors reversing diet-induced obesity in mice

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There is hope that this may lead to advanced therapies for osteoarthritis and may even eliminate the need for surgery in certain settings [7]

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