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Under these conditions, visible harmful effects are not registered and this state is called basal intensity oxidative stress

Results ADH5 limits FA cytotoxicity even in the presence of DNA repair It is still unresolved how FA compromises cellular function, especially in cells proficient for DNA repair, such as those from patients harbouring mutations in the genes coding for FA-metabolizing enzymes

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Upregulation of the NADPH oxidase NOX4 by TGF-beta in hepatocytes is required for its pro-apoptotic activity