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The majority of plasmatic GSH originates from the liver, and for this reason an impairment in hepatic GSH synthesis has a systemic impact on redox balance and oxidative stress (1, 6, 7, 11)

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E., Shalem, O

Results: 197 patients were included in the cohort: 39% alcohol-related cirrhosis, median MELD-Na 14 (range 6-40), 34% Childs Classification C, 65% ascites, 46% encephalopathy

Bairamian D, Sha S, Rolhion N, Sokol H, Dorothe G, Lemere CA, et al

In SAH, AST:ALT was significantly associated at high effect (R=0.539, p=0.031) with LTDH, AUDIT and CONUT in a multivariable regression model