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Boxplot showing decreased GOBP pyroptosis signature scores in primitive compared to committed AML (n = 96 patients)

These agents suppress the Fenton reaction within the mitochondria by reducing the mitochondrial labile iron pool, thereby limiting the formation of OH and preventing mitochondrial lipid peroxidation

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Frequent blood transfusions, exogenous iron consumption, and specific haematological conditions, such as sickle cell anaemia or thalassaemia, are linked to secondary iron overload

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