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Neural and endocrine mechanisms underlying stress-induced suppression of pulsatile LH secretion

Mitochondria are the main production sites of endogenous ROS, so it is feasible to intervene mitochondrial OS in the targeted treatment of AD

Many reports manifest that removal of senescent cells (e.g., via senolytics) or abnormally active transposons (e.g., via inhibitors to LINE1 transposons) from ageing animal models is salutary

Eckmann L, Kagnoff MF, Fierer J: Epithelial cells secrete the chemokine interleukin-8 in response to bacterial entry

Mechanistically, this extract increased eNOS expression while decreasing stress markers, such as nitrotyrosine and GRP78 (Thunuguntla et al., 2024)

Palmieri M.C., Lindermayr C., Bauwe H., Steinhauser C