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Molecular mechanisms of copper in promoting carcinogenesis Copper can promote tumorigenesis by promoting oxidative stress that damages DNA and related molecular structures (152), and contributes to tumor development by promoting angiogenesis, metastasis and cell proliferation through multiple mechanisms: Increased intracellular copper enhances oxidative stress caused by the formation of ROS, leading to elevated intracellular ROS levels (153), DNA damage and activation of oncogenes (59), whereas reduced intracellular copper results in decreased SOD1 activity, nearly complete loss of resistance to oxidative damage and impaired maintenance of normal cellular life activities (121)

While HPV vaccination and screening have reduced cervical cancer incidence, understanding modifiers of HPV persistence and CIN progression remains critical, especially in developing countries

Acknowledgements We acknowledge support from the Imaging Center Essen, G.W

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This is why some people who take acid blockers over the long run have an increased risk of developing C

J neuro-oncology (2018) 136(1):2331