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It is characterized by iron-dependent lipid peroxide damage in mitochondria and a lack of glutathione peroxidase 4 (GPX4) and is distinct from apoptosis, autophagy, and necrosis (Dixon et al., 2012) (Figures 1, 2)

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PMID 30001118

The developing fetus relies entirely on maternal B12 stores, which are actively transported across the placenta

cDCs continuously sample the renal microenvironment

Oxidative stress directly damages lipids, proteins, and DNA in lung tissues, induces an inflammatory response, and also further accelerates the pathological process by inducing iron death in lung tissues [6]