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We show that Nrf2 protein is produced postnatally by the rat choroidal tissue, and translocates in the nucleus of choroidal epithelial cells upon in vivo treatment of rat pups with DMF

Although IFN- suppresses tumor-derived chemokines to inhibit the tumor trafficking of CXCR2 + CD68 + macrophages (associated with M2 polarization) by blocking the CXCL8-CXCR2 axis, thereby counteracting immunosuppression and overcoming therapeutic resistance, dysregulated IFN signaling can promote M2-skewed macrophage polarization, which compromises the efficacy of anti-PD-1/PD-L1 therapies [299]

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& Austin, R

[DOI] [PubMed] [Google Scholar] 119.Zhan H.Q., Najmi M., Lin K., Aluri S., Fiser A., Goldman I.D., Zhao R

doi: 10.1152/physrev.00023.2004