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Here, the inhibition of wounding-induced ROS accumulation significantly inhibited healing in planarian worms by preventing F-actin reorganisation and epithelial cell rearrangements mediated through c-JUN activation at the wound site 122

This protein can create an iron-overloaded environment and lay the foundation for the occurrence of cellular ferroptosis

L., & Magrina, J

In 2000, Ram Kannan asserted that GSH passes through the BBB via Na-dependent GSH transporter in human cerebrovascular endothelial cells (HCBC) and Na-dependent GSH transporter exists in the luminal plasma membrane of HCEC (2000, Brain

Copper and CuNPs have the ability to stimulate apoptosis by caspase-dependent and independent pathways through induction of pro-apoptotic protein (Bax) and suppression of anti-apoptotic protein (Bcl-2) [66, 90, 96], while chrysin activates apoptosis through intrinsic pathways of apoptosis in view of p53 and Bak, caspase-9 and caspase-3 enhancement, and Bcl2 downregulation [96]

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