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Existing research have not compared nutritional "looting" with tumour evolution.Our study constructed risk models to predict the "fate" of tumors and identified crucial molecules that could modify the HCC competitive advantage of "looting" nutrients

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Whitmore LC, Hilkin BM, Goss KL, Wahle EM, Colaizy TT, Boggiatto PM, et al

Here we describe the in vitro and in vivo reconstitution of the entire pathway, starting from the as yet uncharacterized gene products of the carnitine degradation gene operon

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