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Psychiatry 4:e347

The regular vitamin D metabolic profiles illustrated with either wild-type mouse VDR or VDR null mice transgenic for the wild-type human VDR, but not the ligand binding-defective form of the protein, this suggests the essential roles of VDR on VD metabolism 25

Moreover, the lethal phenotype of a homozygous GPx4 knockout makes it difficult to study the physiological role of GPx4 during embryo development

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Excessive ROS accumulation, mitochondrial DNA damage, and impaired mitophagy contribute to compromised oocyte and granulosa cell survival