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Insulin resistance is a feature of T2D, but the primary mechanisms by which it is associated with ferroptosis are increased oxidative stress and lipid peroxidation

Such dysregulation induces oxidative stress and apoptosis in neuronal cells, contributing to neurological disorders associated with HHcy, including age-related macular degeneration, Alzheimers disease, Parkinsons disease, autism, schizophrenia, bipolar disorder, vascular dementia, peripheral neuritis, stroke, multiple sclerosis, epilepsy, and headaches [30, 46, 47, 117]

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