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An additional uncertainty is the specicity of the modication, that is, whether the modi- ed cysteine residue is glutathionylated in vivo, rather than nitrosylated or a target of TRX (or some combina- tion of these modications)

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HDAC inhibition leads to a more permissive chromatin state, enhancing transcription of genes involved in neuronal survival, synaptic plasticity, and memory formation [106]