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Zhang H, Yue X, Chen Z, Liu C, Wu W, Zhang N, et al

We conducted quantitative redox proteomics in primary mouse hepatocytes to identify glutathione pool-sensitive S-glutathionylated proteins and found that S-glutathionylation is required to maintain the function of ADP-ribosylation factor 6 (ARF6)

Based on the conclusion of transcriptomic data, derived from the INH-treated HepG2 human liver cell line, we identified OS-related genes and pathways affected by INH treatment, which are essential for maintaining cellular redox balance

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Others contain multiple doses or need to be refilled

Sections were rehydrated in PBS and fixed in 4% paraformaldehyde at RT for 10 min