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We will later discuss the evidence of microbes in the pathogenesis of AD in patients, but first it is important to understand the role of tau protein, another prominent player in the pathogenesis of AD as this protein also bears similarities with A, despite different functional roles

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[5] Through gene regulation, estradiol promotes vaginal epithelial maturation, supports collagen synthesis within skin, and contributes to nitric-oxide-mediated vasodilation, collectively providing mechanistic explanations for clinical benefits such as improved vulvovaginal comfort and reduction in hot-flash frequency

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