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158,159,160 Recent research shows that hepatocytes release cholesterol and C-X-C motif chemokine ligand 1 (CXCL1), drawing neutrophils from the bone marrow to the injured liver, where they subsequently secrete HGF to enhance hepatocyte proliferation and support liver regeneration

For instance, research shows that luteolin and kaempferol can reduce fibroblast proliferation and myofibroblast (MYF) differentiation by activating the AMPK/PPAR- pathway

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This creates feed-forward cycles that exacerbate cuproptosis: mtROS oxidize cytosolic GSH, releasing bound Cu, and may upregulate the copper importer CTR1 via pathways such as NF-B, increasing copper influx [65]

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