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Further studies revealed that mechanistic studies confirmed that DHODH could only perform its ferroptosis inhibitory function when localized in mitochondria and that this function was dependent on its enzymatic activity

SOD1 transcriptional and posttranscriptional regulation and its potential implications in ALS

doi: 10.3390/molecules28030982 82 Hyun-OhLJi-WeonCJeong-HoBJae-HyeonOSang-ChoonLChang-KugK

Conclusion and future perspectives MASLD has a globally increasing prevalence with the potential to progress to its more severe form, MASH, and is an independent risk factor for cardiometabolic disorders