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As such, the overall structure of rozanolixizumab differs from that of other full-length FcRn antagonist mAbs in active development, most of which are of the IgG1 subclass (Fig

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Biochem Pharmacol, 33: 1005-1013

While 100 nM MitoQ only minimally increased DPI-induced cell death ( Figure 1C ), higher MitoQ concentrations significantly enhanced DPI-mediated cell toxicity as measured by an increased PI uptake after 24h, in a MYCN-dependent fashion ( Figure 1E )

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