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Reduction of Inflammatory Signaling Cartalax has been shown to reduce inflammatory activity within cartilage tissue by: Suppressing pro-inflammatory cytokine signaling Reducing matrix metalloproteinase (MMP) activity Supporting a more balanced inflammatory environment This is particularly important in osteoarthritis, where inflammation drives progressive cartilage loss

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Type-I IFN: Type-I IFN facilitates dendritic cell (DC) maturation, enhances autoimmune T cell survival, and activates autoantibody-producing B cells, primarily through sustained IFN- production, autophosphorylation, and activation of the IFN- receptor (IFNAR)-associated Janus kinases (JAKs) and signal transducers and activators of transcription (STATs) (87, 88)