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Homodimerization is a prerequisite for TBK1 activation, and mutants EE (double site in E355R/E448R) and HIF (triple site in H459E/I466E/F470E) abolish TBK1 dimerization [23, 24]

This most likely reflects the potential toxicity of this compound in relatively high levels, observed under in vitro and in vivo conditions (Yang et al., 2000

10.1158/0008-5472.CAN-18-0440 90 StockwellB

In addition, serine metabolism to cystathionine, cysteine, and GSH as traced with 13 C 3 -serine showed no detectable GSH biosynthesis from the TSS pathway (Supplementary Fig

it is naturally occurring, nontoxic, and is active at a very low nanomolar concentration.11 Another piece of research described GHK-Cu as a safe, inexpensive, extensively studied compound.3 These conclusions primarily apply to topical GHK-Cu, but even with injectables, the lack of serious adverse events in animal subjects points to a general tolerability regardless of the route of administration