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TQ has a quinone molecular structure for redox activity and the infinite capability to cover all physiological barriers so that it has easy access to subcellular compartments, which affects radical scavenging (101)

Combined inhibition synergistically and irreversibly reactivates APC/C CDH1 and induces the degradation of PIN1 and other mitotic proteins, leading to irreversible cell cycle exit, which translates into synergistic anti-tumor activity against triple-negative breast cancer both in immune-compromised and -competent and/or RB -deficient or -proficient mouse models

The conserved kinases CDK-1, GSK-3, KIN-19, and MBK-2 promote OMA-1 destruction to regulate the oocyte-to-embryo transition in C

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