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Senolytic agents focus on clearing the burden of senescent cells, directly addressing the source of cellular dysfunction, while senomorphics target the pro-inflammatory and tissue-degrading effects of the SASP, which result from the senescence process

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A 4-amino acid synthetic tetrapeptide modeled on pineal gland epithalamin that activates telomerase and modulates circadian gene expression in preclinical and early human studies

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Pirin promotes the progression of non-small-cell lung cancer by increasing ODC1 to suppress autophagy

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