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Cammann, V

Yamanishi and Tuboi, 1981), and Ida et al

MNA cells rely on an elevated antioxidant response through the Xc-/glutathione (GSH) pathway to scavenge lipid hydroperoxides, a by-product formed from excess H 2 O 2 (11, 12)

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Prospectively, modulation of ferroptosis may be achieved by targeting specific enzymes within this pathway, either to promote cell death in cancer cells or to prevent excessive damage in normal cells