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Importantly, these effects may be mediated not only through the development of mental illness but also through intermediate disruptions in emotional development, such as attachment insecurity (32, 41, 42)

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As positive and negative controls, respectively, we included the glycolytic targets of HIF1, established previously to be regulated downstream of mTORC1 (Duvel et al., 2010), and the mitochondrial tRNA synthetases, not believed to be regulated by ATF4

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