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Disclosures: Qian Xue: Nothing to Disclose, Liang Deng: Nothing to Disclose, Jinqiu Zhao: Nothing to Disclose, Fengling Peng: Nothing to Disclose, Youping Zhou: Nothing to Disclose, Li Zhong: Nothing to Disclose, Lu Huang: Nothing to Disclose, Shan Li: Nothing to Disclose, Chunhong Guo: Nothing to Disclose 2007 HUC-MSCS EXOSOME-DERIVED MIR-100-5P ALLEVIATES LIVER FIBROSIS BY INDUCING AUTOPHAGIC DEATH OF HEPATIC STELLATE CELLS yonghong wang 1 Menglan Wang 1 Yachao Tao 1 dongbo wu 1 enqiang Chen 1 hong Tang 1 , 1 West China hospital, Sichuan University Background: The incidence of end-stage liver disease is very high in the late stage of liver fibrosis,so early treatment of liver fibrosis is of great significance

In addition, the decrease of KGDHC immunostaining was roughly related to the severity of degeneration

Nagapan, T.S ., Lim, W.N., Basri, D.F

doi: 10.1002/jcb.29258 70 LuTXRothenbergME

Glutathione and immune system support: Mechanisms and benefits in aging populations

In a second experiment, incubation of the SetCys peptide with a peptide thioester in the presence of MPAA furnished a ligated peptide featuring an internal SetCys residue (Fig