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Genetic variants in nutrient metabolism pathwayssuch as MTHFR polymorphisms affecting folate and B12 cycling, or PEMT variants influencing choline requirementsmay inform whether you are at higher risk for deficiency or responsiveness to lipotropic support

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The findings in mouse models of SOD1 ALS show an increase of complement activators (C1qB, C4, C3, C5, and CD88) and decrease in complement regulators (CD55, CD59a) during disease progression in spinal-cord-tissue supported investigations of complement inhibitors in ALS [256]

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