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The autophagic degradation of BMAL1 and CLOCK, termed Clockophagy, is a recently identified pathway for ferroptosis [93], which could intersect with BD pathogenesis

Such targeting has the potential to make targeted drug delivery possible, allowing therapeutic factors that can damage the healthy part of the brain to be delivered only to the diseased region

In doing this, the potential of therapies that have a significant impact on a small subset is lost within the larger sample population

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Structural and functional abnormalities in the amygdala, hippocampus and medial prefrontal cortex underlie persistent, overgeneralized fear responses (Harnett et al., 2020)