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PfGrx C32S/C88S (SSG) catalyzed the oxidation of roGFP2 via a monothiol mechanism and reacted with all roGFP2 variants 1.5 to 3 times faster than PfGrx C88S (SSG) (Fig

2.12 Statistical analysis The data was analyzed using the SPSS software system (version 22)

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Correlation heatmap of TurboID interactomes comparing HMGB1/2-TurboID groups (K-Hcy modification) with background controls (TurboID-only)

The putative mechanisms underlying altered body composition with CLA include reduced lipogenesis and increased lipolysis, increased expression of genes that interfere with the maturation of adipocytes [86], increased fat oxidation via elevated activity of carnitine-palmitoil-transferase-1 (CAT-1), reduced activity of lipoprotein lipase, inhibition of adipocyte differentiation, and increased activation of apoptotic pathways in adipose tissue [21, 87, 88]