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Initial screening of the in vivo activity of the LiTE and Albu-LiTE-HB was investigated in an immunodeficient C57BL/6 RAG1 knockout mouse model inoculated with a 2:1 mixture of HT-29 cells and human PBMCs using multiple (8) drug doses to compensate for the short half-life of the unfused LiTE (Supplementary Fig

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110,111 Nonhomologous end joining plays a vital role in the repair of double-strand breaks, particularly in cells that are not actively replicating, by directly connecting the damaged ends of DNA