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used angiotensin (Ang) II injection to simulate a hypertension-mediated cardiac remodeling model and found that ferroptosis occurred and that the protein expression of GPX4 and xCT was downregulated in the myocardium

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Early identification and treatment can help mitigate some of these adverse metabolic effects

Conclusions: We do not report altered GSH concentrations in the ACC or DLPFC in PTSD, however, systemic MMPs and MPO might be implicated in central processes and progression of PTSD

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CS exposure depleted ELF GSH levels by 4 hours from 100 M to 25 M, which was roughly the level where it remained until the end of the CS exposure (Figure 3A)