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In HCECs applied with desiccating stress, the CEC ferroptosis was triggered by the aggregation of autophagy impairment-derived sequestosome 1 (SQSTM1), the overexpression of which fostered ferroptotic changes and substantially elevated ACSL4 (124)

Ebrahimi-Mameghani M, Jamali H, Mahdavi R, Kakaei F, Abedi R, Kabir-Mamdooh B

In addition, preliminary evidence suggests that vitamin C can accumulate in neutrophils to an extent

GHK-Cu has been shown to accelerate wound closure, reduce scarring, and promote the formation of new blood vessels (angiogenesis) crucial for tissue repair

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The CAT enzyme activity was evaluated from the decline in absorbance at 240 nm due to the loss of H 2 O 2