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Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathwaysparticularly those involving proline, arginine, glutamine, and tryptophandrive collagen deposition, fibroblast activation, and disease progression

Funding This work was supported by a grant from the Health Science and Technology Foundation of Zhenjiang (SH2023024)

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