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10.1016/j.celrep.2021.108736 38 GarrisonG

For example, KRAS-G12C harbours a unique cysteine residue that enables covalent binding by allele-specific inhibitors,24 a feature absent in G12D and G12V, which limits their druggability and necessitates alternative targeting strategies

A plausible interpretation of the results is that AtGrxS15 was able to react with GSSG and that glutathionylated AtGrxS15 subsequently transferred its glutathione moiety to reduced roGFP2

The novel triterpenoid CDDO-Me suppresses MAPK pathways and promotes p38 activation in acute myeloid leukemia cells

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