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Mechanisms responsible for the subsequent development of LC include virus-specific pathomechanisms described above, aberration of immune reaction and development of OS and inflammation-mediated cellular damage (Figure 2) as a reaction to acute infection, and expected sequelae of post-critical illness (67)

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Release of glial tissue-specific proteins after acute stroke: A comparative analysis of serum concentrations of protein S-100B and glial fibrillary acidic protein

Long work hours, disrupted sleep patterns, chronic stress and environmental toxins all take a toll

Movement and Core Stimulation Sedentary behavior worsens constipation

Incidence of post-cataract endophthalmitis at Aravind Eye Hospital: outcomes of more than 42,000 consecutive cases using standardized sterilization and prophylaxis protocols