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Similar to the reduction in GSH-PX activity, the disruption of the PRX/Trx-mediated ROS scavenging through the depletion of Trx2 neonatal rat ventricular cardiomyocytes causes cardiomyocyte hypertrophy 10 , and mice with cardiomyocyte-specific deletion of Trx2 develop dilated cardiomyopathy with a severe decline in cardiac contractility and early mortality 11

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Chaudhary et al., 2022

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