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Studies in mice showed that colistin toxicity is associated with the activation of (i) the mitochondrial apoptotic pathway, (ii) the death receptor pathway, and (iii) the endoplasmic reticulum pathway 20,21

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In addition to endocrinology data, such as levels of testosterone, luteinizing hormone, androstenedione, and 5-dihydrotestosterone, which can vary widely in AIS individuals, the most reliable approach is the sequencing of each individual androgen receptor exon and the flanking intron sequences