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To ensure the proper function of SLC7A11 in maintaining redox homeostasis, the expression and activity of SLC7A11 is strictly regulated by a variety of mechanisms, including transcriptional and epigenetic regulator-mediated transcriptional regulation, as well as post-transcriptional regulatory mechanisms

Within the mitochondria, adenosylcobalamin serves as an absolute, non-negotiable requirement for the function of an enzyme called methylmalonyl-CoA mutase (MUT)