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Evidence from animal studies suggests that the effects of GCs on LTP and memory follow an inverted U-shaped dose-response relationship, with moderate physiological levels supporting synaptic plasticity and memory consolidation, whereas both insufficient and excessive GC exposure are associated with impairment (Pavlides et al., 2002

13 Yoshinori Ohsumis identification of autophagy-related genes in yeast during the 1990s defined the molecular machinery of autophagy, 14,15 while later work highlighted selective mitochondrial autophagy (mitophagy) as essential for mitochondrial quality control and cellular survival 16,17 (Fig

In addition, one hallmark of neurodegenerative processes is the disruption of the inflammatory response [3]

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Additionally, insulin resistance induces hyperinsulinemia, which further suppresses SHBG production, exacerbating reproductive dysfunction