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Description

In vitro assessments of cytotoxicity and cellular uptake revealed the efficient release of DOX in hypoxia environments upon azo bond cleavage, leading to higher cytotoxicity than in normal conditions

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I did not do anything else extra in terms of recovery that I normally would do

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Nature 422 , 130131 (2003)

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Seizure-induced changes in mitochondrial redox status

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Read more about the RAAS here

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Loss of cGAS abolished senescence phenotypes and senescence-associated -galactosidase (SA--Gal) activity, suggesting that cGAS is essential for cellular senescence (Yang et al., 2017)

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