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Binding of cGAS to Beclin-1 leads to the release of Rubicon, thereby enabling autophagic degradation of cytosolic dsDNA and resulting in decreased cGAS-STING signaling ( Figure 3 )

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Cardiovascular and Metabolic Studies Preliminary studies suggest that BPC-157 may contribute to cardiovascular healing by enhancing endothelial function and mitigating oxidative stress

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