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Mechanistic Interpretation The study identifies a dual immunosuppressive role for IGF-1 in prostate cancer: Antigen presentation impairment: IGF-1 disrupts peptide transport and processing (via TAP1/2, ERAP1, 2M downregulation), reducing surface MHC-I expression and impairing CD8 T-cell recognition

Here, we outline how these pathways from a crucial metabolic circuit that is a key determinant of T cell function ( Figure 2 )

Furthermore, neuron autophagy was activated in the presence of this compound, which increased histone acetylation

In other words, they prevent neuronal hyperactivity

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doi:10.1016/0014-2999(86)90596-0