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2011 in the Journal of Applied Physiology characterized BPC-157's effects on tendon fibroblast outgrowth through increased FAK phosphorylation and downstream paxillin activation, with measurable effects on cell migration rate and tendon-derived fibroblast survival after mechanical injury

In parallel, the higher but transient P53 expression in Gstp1/2 / mice could contribute to the maintenance of the redox homeostasis and cell survival as well as the early increase in GSTM1 protein levels that blocks the TRAF2/ASK1 signaling pathway