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Brain mitochondria as potential therapeutic targets for managing hepatic encephalopathy

Histamine N-Methyltransferase in the Brain. International Journal of Molecular Sciences, vol

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10,268 Additionally, alternative activation pathways, such as PKC-mediated EGFR-mTOR signaling, indicate that multiple regulatory inputs sustain mTOR activity in GBM

A recent study found that genetic DAO deficiency contributed to histamine intolerance in 79% of patients.[ref] (Members will see their DAO genotypes below in the genotype report.) Historical note: Excess histamine is the cause of scombroid poisoning a type of food poisoning that occurs after consuming fish that is not fresh

Data availability: The datasets generated during and/or analysed during the current study are available from the corresponding authors on reasonable request