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Overall, these data highlight the pivotal role of the ABCC4-cAMP-Epac2/Rap1a signaling pathway in modulating surface LDLR availability and LDL-C uptake in hepatocytes

Following long-term exposure to UV-A (up to 87 weeks), insoluble protein levels rose to 46% higher than controls (153), which is similar to the accumulation of insoluble proteins observed in ARN cataracts in humans (124, 154)